Endothelium-Dependent Hyperpolarizations
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Endothelium-Dependent Hyperpolarizations

Endothelium-Dependent Hyperpolarizations


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About the Book

The existence of endothelium-dependent hyperpolarization and endothelium-derived hyperpolarizing factor was established during 1997 at an international symposium held in near Paris. This further volume reinforces the concept that EDHF, as opposed to nitric oxide (NO), is of particular importance in smaller blood vessels and, in addition, underlines the multiplicity of other factors involved. Many candidate mentors are discussed, including some new, sometimes surprising ones and the actions of EDHF on vascular vascular smooth muscle cells are detailed, almost down to the molecular level. Further chapters enlarge our knowledge of the physiological and pathological roles of endothelium-dependent hyperpolarizations, and the book concludes with reports on the still limited but crucial knowledge of the contribution of EDHF in human blood vessels in health and disease.

Table of Contents:
Endothelium-dependent hyperpolarization cannot be explained by electrical coupling between the endothelial and the smooth cells in muscular arteries; epoxyeicosatrienoic acids as endothelium-derived hyperpolarization factors in coronary arteries - potassium channel activation through endogenous ADP - ribosylation of Gs; phospholipid-derived epoxyeicosatrienoic acids mediate the relaxation's attributed to endothelium-derived hyperpolarizing factor; hyperpolarizing factors and membrane potential in cerebral arterioles - a role for astrocytes and cytochrome P450 genes; K+ is an endothelium-derived hyperpolarizing factor - an alternative working hypothesis; hyperpolarization and relaxation of vascular smooth muscle to endothelium-derived nitric oxide; endothelium-dependent responses in endothelial nitric oxide synthase knockout mice - contribution of NO, EDHF and products of cyclooxygenases; SR141716A does not inhibit non-nitric oxide, non-prostanoid mediated relaxations to ethanol and cyclopiazonic acid in the isolated porcine pulmonary artery; cannabinoid receptors are present in arterial smooth muscle, are coupled to the inhibition of adenylate cyclase, but are not involved in vasorelaxation; potassium channels involved in hyperpolarization and relaxation induced by endothelium-derived hyperpolarizing factor in rat mesenteric arteries; the role of nonselective cation current in regulation of membrane potential in vascular smooth muscle cells; diminished cyclic GMP - and hyperpolarization-dependent ralaxation's to acetylcholine in rabbit carotid arteries with a neointima; contribution of nitric oxide and endothelium-derived hyperpolarizing factor to the relaxation evoked by bradykinin in porcine coronary arteries four weeks after removal of the endothelium; role of endothelium-derived hyperpolarizing factor in relaxation of pregnant rat uterine artery by corticotrophin-releasing factor; endothelium-derived hyperpolarizing factor in omental arteries from normotensive pregnant women; mechanisms of vasodilation in human conduit arteries; vasodilation to bradykinin in mediated by an endothelium-derived hyperpolarizing factor essential in hypertensive patients; importance of endothelium-derived hyperpolarizing factor in human arteries.


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Product Details
  • ISBN-13: 9789057024924
  • Publisher: Taylor & Francis Ltd
  • Binding: Hardback
  • Language: English
  • No of Pages: 468
  • Weight: 1207 gr
  • ISBN-10: 9057024926
  • Publisher Date: 31 Oct 2000
  • Height: 254 mm
  • No of Pages: 468
  • Returnable: N
  • Width: 178 mm


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