Apoptose
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Home > Art, Film & Photography > Apoptose: Survivin, P53, Apoptose-Inhibitor, Bcl-2, Perforin, Hayflick-Grenze, Bcl-2-Antagonist-Of-Cell-Death
Apoptose: Survivin, P53, Apoptose-Inhibitor, Bcl-2, Perforin, Hayflick-Grenze, Bcl-2-Antagonist-Of-Cell-Death

Apoptose: Survivin, P53, Apoptose-Inhibitor, Bcl-2, Perforin, Hayflick-Grenze, Bcl-2-Antagonist-Of-Cell-Death


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About the Book

Aus Wikipedia. Nicht dargestellt. Auszug: Apoptosis (pronounced or ) is the process of programmed cell death (PCD) that may occur in multicellular organisms. Biochemical events lead to characteristic cell changes (morphology) and death. These changes include blebbing, loss of cell membrane asymmetry and attachment, cell shrinkage, nuclear fragmentation, chromatin condensation, and chromosomal DNA fragmentation. (See also Apoptosis DNA fragmentation.) Apoptosis differs from necrosis, in which the cellular debris can damage the organism. Histologic cross section of embryonic foot of mouse (Mus musculus) in 15.5 day of its development. There are still cells between fingers. (Full development of mouse lasts 27 days.) (Compare this image with image of leg of mouse.)In contrast to necrosis, which is a form of traumatic cell death that results from acute cellular injury, apoptosis, in general, confers advantages during an organism's life cycle. For example, the differentiation of fingers and toes in a developing human embryo occurs because cells between the fingers apoptose; the result is that the digits are separate. Between 50 and 70 billion cells die each day due to apoptosis in the average human adult. For an average child between the ages of 8 and 14, approximately 20 billion to 30 billion cells die a day. Research in and around apoptosis has increased substantially since the early 1990s. In addition to its importance as a biological phenomenon, defective apoptotic processes have been implicated in an extensive variety of diseases. Excessive apoptosis causes atrophy, such as in ischemic damage, whereas an insufficient amount results in uncontrolled cell proliferation, such as cancer. A German scientist Carl Vogt was first to describe the principle of apoptosis in 1842. In 1885, anatomist Walther Flemming delivered a more precise description of the process of programmed cell death. However, it was not until 1965 that the topic was ...http: //booksllc.net/?l=d


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Product Details
  • ISBN-13: 9781158760749
  • Publisher: Books LLC
  • Publisher Imprint: Books LLC
  • Height: 152 mm
  • No of Pages: 42
  • Spine Width: 3 mm
  • Weight: 73 gr
  • ISBN-10: 1158760744
  • Publisher Date: 22 Jul 2010
  • Binding: Paperback
  • Language: English
  • Returnable: N
  • Sub Title: Survivin, P53, Apoptose-Inhibitor, Bcl-2, Perforin, Hayflick-Grenze, Bcl-2-Antagonist-Of-Cell-Death
  • Width: 229 mm


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