About the Book
Please note that the content of this book primarily consists of articles available from Wikipedia or other free sources online. Pages: 64. Chapters: Cell cycle, Meiosis, Cell division, Exocytosis, Endocytosis, Necrosis, Chromosomal crossover, Cellular differentiation, Genetic recombination, DNA repair, Biochemical switches in the cell cycle, Cell growth, Site-specific recombination, Autophagy, Septins, Cell migration, Endoplasmic Reticulum Associated Protein Degradation, Intraflagellar transport, Cap formation, Endocytic cycle, Parasexual cycle, Axon terminal, Phagocytosis, CDK7 pathway, Dynamin, Stringent response, Passive transport, Secretory pathway, Receptor-mediated endocytosis, Efferocytosis, Autolysis, Endoexocytosis, Formins, Nemosis, Coagulative necrosis, Necrobiology, Dentinogenesis, Trans-endocytosis, Bulk endocytosis, Afterhyperpolarization, Autophagin, Pinocytosis, Cytoplasmic streaming, Rap6, Pyknosis, Mitotic crossover, Invagination, Karyolysis, Autophagy database, Density-dependent inhibition, Potocytosis, Transcytosis, Histone methylation, Leptotene stage, Meiome, Necrobiosis, Non-specific, adsorptive pinocytosis, Filamentation, Karyorrhexis, Residual body, Branch migration, Fungating lesion, S phase index, Synizesis, Ectopic recombination, Interkinesis. Excerpt: DNA repair refers to a collection of processes by which a cell identifies and corrects damage to the DNA molecules that encode its genome. In human cells, both normal metabolic activities and environmental factors such as UV light and radiation can cause DNA damage, resulting in as many as 1 million individual molecular lesions per cell per day. Many of these lesions cause structural damage to the DNA molecule and can alter or eliminate the cell's ability to transcribe the gene that the affected DNA encodes. Other lesions induce potentially harmful mutations in the cell's genome, which affect the survival of its daughter cells after it undergoes mitosis. As a consequence, the DNA repair...