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Mitochondria: Mitochondrial Permeability Transition, Programmed Cell Death, Translocase of the Outer Membrane

Mitochondria: Mitochondrial Permeability Transition, Programmed Cell Death, Translocase of the Outer Membrane


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Chapters: Mitochondrial Permeability Transition, Programmed Cell Death, Translocase of the Outer Membrane, Translocase of the Inner Membrane, Mitochondrial Membrane Transport Protein, Tim/tom Complex, Kinetoplast, Oncocyte, Bcl-Xl. Source: Wikipedia. Pages: 41. Not illustrated. Free updates online. Purchase includes a free trial membership in the publisher's book club where you can select from more than a million books without charge. Excerpt: Mitochondrial permeability transition, or MPT, is an increase in the permeability of the mitochondrial membranes to molecules of less than 1500 Daltons in molecular weight. MPT results from opening of mitochondrial permeability transition pores, also known as the MPT pores or MPTP. The MPT pore is a protein pore that is formed in the membranes of mitochondria under certain pathological conditions such as traumatic brain injury and stroke. Induction of the permeability transition pore can lead to mitochondrial swelling and cell death and plays an important role in some types of apoptosis. The MPTP was proposed by Haworth and Hunter in 1979 and has since been found to be involved in, among other things, neurodegeneration, a process that results in damage and death of neurons. MPT is frequently studied in liver cells, which have especially large numbers of mitochondria. MPT is one of the major causes of cell death in a variety of conditions. For example, it is key in cell death in excitotoxicity, in which overactivation of glutamate receptors causes excessive calcium entry into the cell. MPT also appears to play a key role in damage caused by ischemia, as occurs in a heart attack and stroke. However, research has shown that the MPT pore remains closed during ischemia, but opens once the tissues are reperfused with blood after the ischemic period, playing a role in reperfusion injury. MPT is also thought to underlie the cell death induced by Reye's syndrome, since chemicals that can cause...More: http://booksllc.net/?id=4135795


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Product Details
  • ISBN-13: 9781156220443
  • Publisher: Books LLC
  • Publisher Imprint: Books LLC
  • Height: 152 mm
  • Sub Title: Mitochondrial Permeability Transition, Programmed Cell Death, Translocase of the Outer Membrane
  • Width: 229 mm
  • ISBN-10: 1156220440
  • Publisher Date: 15 Sep 2010
  • Binding: Paperback
  • Spine Width: 3 mm
  • Weight: 73 gr


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